The Effect of Human Peptide GHK Relevant to
Nervous System Function and Cognitive Decline
Brain Sciences (2017)
New Data of the Cosmeceutical and TriPeptide GHK
SOFW Journal (2015)
GHK-Cu May Prevent Oxidative Stress in Skin
by Regulating Copper and Modifying Expression of
Numerous Antioxidant Genes Cosmetics (2015)
GHK Peptide as a Natural Modulator of
Multiple Cellular Pathways in Skin Regeneration (2015)
GHK, the Human Skin Remodeling Peptide Induces Anti-Cancer
Expression and DNA Repair Analytical Oncology (2014)
GHK and DNA: Resetting the Human Genome to Health
BioMed Research International (2014)
Avoid Buying Fake Copper Peptides Dangerous








Tissue Remodeling Copper Peptides Induce Regeneration of Diverse Organs

Effect of GHK in Mammals

I discovered GHK-Cu in human plasma during experiments that tried to reverse certain effects of aging in animals. Since then, GHK-Cu and certain other copper-peptides, have been found to accelerate the regeneration and repair of many types of mammalian tissues. Studies, at numerous universities and research institutes, have found copper-peptides to:       

- Accelerate wound repair (humans, mice, rats, guinea pigs, pigs, dogs)       

- Increase skin re-epithelialization (humans, animals)       

- Reverse aging effects on skin (humans) - thickens skin, reduces small and large wrinkles, reduces blotchiness and blemishes, improves elasticity, and increases keratinocyte and fibroblast production, thickens the subcutaneous fat layer       

- Improve skin graft transplant success (pigs)       

- Improve hair transplant success (humans)       

- GHK analogs with fatty residue analogs increase hair follicle size, stimulate hair growth and reduce hair loss (humans, mice, rats). Increases hair growth after chemotherapy (rats)       

- Stimulate bone healing (guinea pigs, pigs, rabbits)       

- Heal injured intestinal linings (humans, rats)       

- Heal stomach ulcers (rats)       

- Block oxidative injury in tissues.

- Restore normal function in lives damaged by toxins (rats)

- Have profound anti-inflammatory actions (reduces release of oxidizing iron from ferritin, TNF-alpha, TGF-beta-1, IL-1). The actions on TNF-alpha (tumor necrosis factor alpha) are especially important since it is considered to the the "master inflammatory molecule" that plays a causative role in chronic inflammatory conditions (rheumatoid arthritis, Crohn's disease, psoriasis, psoriatic arthritis, ankylosing spondylitis, ulcerative colitis, acute alcoholic hepatitis) and possibily atherosclerosis, Alzheimer’s disease, and neuropathies. Drugs that block TNF-alpha also block the other cytokines involved in the inflammation.

Questions or Advice?

Email Dr. Loren Pickart at drlorenpickart@gmail.com

Alternate Email: ghkcopperpeptides@gmail.com

Call us at 1-800-405-1912 Monday through Friday (8 am to 6 pm) PST